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The value of GSH as an immunopharmacologic agent is that it can finetune immune response in a disease-dependent and redox state-dependent manner which differs from typical broad-effect immunosuppressive treatments (Diotallevi et al., 2017) (Figure 3)

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For example, lactate derived from CAFs negatively regulates the Hippo signaling pathway via the DLG5/CUL3/MST1 axis, promoting YAP1 nuclear localization and enhancing CSC-like phenotypes

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