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However, unlike necrosis, pyroptosis was initially described as a type of programmed cell death that, rather than being quiet, like apoptosis, triggers a robust inflammatory response, including the recruitment of immune cells to the site of death (Cookson and Brennan, 2001)

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[7] In other words, by inhibiting this interaction, FOXO4-DRI might enhance the organism’s ability to clear these cells without significantly impacting other functions of FOXO4

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