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All things considered, mitochondrial dysfunction contributes to abundant cardiac pathologies: ischemia-related disorders, atherosclerosis, heart failure, and metabolic disease [6,7,8,9,10,11,12,13,14,15,16,17,19,22,23,24], whereby its pharmacological restoration emerges as an intriguing therapeutic avenue
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The peptide appears to work by normalizing sleep architecture rather than acutely sedating, which explains the variable onset
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