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Hypoxia not only affects the function of innate immune cells (such as macrophages, neutrophils, dendritic cells, and natural killer cells) but also regulates the differentiation of adaptive immune cells (such as T cells and B cells), which promotes the inflammatory state of RA disease and affects the pathological process of RA

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Since growth hormone signaling drives cellular proliferation and protein synthesis in fibroblasts, this amplification creates a stronger repair response