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Cytosolic/nuclear fractionation immunoblots of HMGB1/2-GFP wild-type (WT) and lysine-to-arginine mutants in HTL-treated HCMECs
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This discovery suggests that these molecules may exacerbate AV by promoting the cell cycle, aggravate the inflammatory response, and further activating the immune reaction, thereby contributing to the onset and worsening of AV
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